An angina attack can be initiated by one of several processes. Walking or other exercise releases catecholamines which have the ability to divert blood from other vascular beds to the heart, brain and muscles. At the same time blood vessels dilate causing blood pressure to decrease. When insufficient ATP is being synthesised or the blood fails to supply sufficient oxygen the symptoms of angina are experienced. Because the synthesis of angiotensinogen II requires renin and the release of renin is inhibited by the catecholamines which circulate in blood during exercise, this type of angina does not involve angiotensinogen and can be controlled by stopping physical activity. Alternatively adequate control can be achieved by the use of coro-nitro to dilate coronary arteries sufficiently to enable physical activity to be resumed at a decreased rate.

When the flow of blood through the arteries is restricted by the presence of atheroslerotic plaque the flow can be increased after using up to six grams of vitamin C a day to dissolve the plaque. An additional supplement of unsaturated fats ensures that low density lipoprotein contains cholesterol which is combined with oleate and that high density lipoprotein contains sufficient polyunsaturated fats to facilitate the scavenging of free cholesterol from the blood. Cholesteryloleate is transported by low density lipoprotein to the fat reserves but free cholesterol is transported by high density lipoprotein to the liver for acidification and excretion in bile.

For heart muscle to be enabled to produce maximum quantities of ATP it needs to contain sufficient minerals and vitamins as well as oxygen. If deficiencies of vitamins B3 and B6, folic acid, vitamin D and magnesium are present they need to be corrected, as do deficiencies of iodine and selenium to ensure an adequate supply of triiodothyronine (T3). Without a sufficient supply of these nutrients and sufficient T3 the correct basal rate of ATP synthesis cannot be achieved and all metabolic processes become less efficient.  Failure to synthesise sufficient ATP adversely affects the activity of calcium ATPase so that the intracellular concentration of calcium ions increases. This causes the smooth muscle around the arteries to contract so that in the case of the coronary arteries less oxygen transporting blood is enabled to suffuse heart muscle.

One group of drugs which is commonly prescribed to those who suffer from hypercholesterolaemia may adversely affect ATP synthesis by preventing ubiquinone synthesis. Drugs which block the action of angiotensinogen II also prevent the normal release of aldosterone. The consequent loss of sodium and chloride in urine may decrease blood pressure, but when too much is lost sodium-potassium ATPase activity decreases and body temperature falls. At the same time the loss of chloride can decrease the ability to produce gastric acid and digest food.

Physical inactivity when accompanied by exposure to weak electromagnetic fields released from TV sets, low energy lightbulbs, etc favours angiotensinogen II synthesis as soon as the exposure stops. Because the low energy light bulbs contain mercury they emit electromagnetism even when out of use and not switched on. The same may be true of other pieces of electrical apparatus. What appears to happen during exposure to electromagnetic fields is that homeostatic factors prevent smooth muscles contracting but as soon as the subject moves out of the field there is a rapid fall in blood pressure which is sufficient to enable renin to be released and angiotensinogen II to be produced. In normally healthy people the amount of angiotensinogen II formed is very limited because blood pressure is at once restored, but in those who have had a stroke or have arteries which are occluded by plaque the response may be delayed until more is produced. In this situation when the response develops it is accompanied by an angina attack. Attacks of this kind are very unpleasant and can follow soon after leaving a TV set to go to bed. They can also follow a short sleep. In both cases they become very painful unless coro-nitro is used as soon as they have started.

In addition to inducing homeostatic effects weak electromagnetic fields can decrease the potency of T3 by causing its molecules to adopt the form in which the iodine atom on the benzene ring is twisted into the proximal position in relation to the rest of the molecule. This is achieved by the rotation of the benzene ring upon the oxygen atom attaching it to the rest of the molecule. When the iodine atom on the benzene ring is in the distal position T3 has greater activity than when it is in the proximal position.

It has been found through personal experience that a bedside lamp with a mercury vapour lamp can induce angina attacks at night following a visit to the toilet and that these attacks can be avoided either by removing the bulb or by wearing a magnetic bracelet. When the bulb is left in place and the bracelet removed, a deterioration in mood follows about one day later but improves soon after the bracelet has been replaced. Mood is unaffected by taking the bracelet off after the bulb has been removed. When away from home and exposed to two mercury bulbs after a period during which there had been no exposure to an electromagnetic field and when the bracelet was not being worn there was no effect during the first night. During the second night severe problems were experienced which could not be alleviated by using coro-nitro, but thereafter sleep was normal because the mercury bulbs had been removed.

About a day is needed to enable good T3 to be replaced in cell structures by bad T3, and vice versa, and the ratio of good to bad T3 can be increased by wearing a magnetic bracelet. Presumably there is a normal ratio of good to bad T3 in plasma and by altering the ratio it is possible to influence susceptibility to problems such as asthma, seasonal afferent depression, and manic depression. Presumably good and bad T3 differ in the potency of the feedback effects they exert upon the pituitary gland and its production of thyroid releasing hormone. According to a textbook the half-life of T3 in plasma is about one day. This seems to explain the delayed effects which were experienced as one type of T3 replaced the other.

It was unexpected to discover that the incidence of angina attacks could be decreased by improving the rate of synthesis of ATP as the maximum rate of ATP synthesis usually depends upon the supply of oxygen. It follows, at least in some cases, that the primary cause of angina is the failure to synthesise sufficient ATP and not oxygen deficiency. This confirms that strokes and heart disease when associated with the presence of atherosclerotic plaque are diseases which are caused by nutritional deficiencies and cannot be effectively treated solely by drugs.

Mercury vapour lamps are a health hazard and should never be used in bedrooms or sitting rooms where individuals habitually spend much of their time. Children who spend much of their time watching television risk mood problems as well as obesity. Obesity develops because bad T3 lowers the rate of metabolism and lowers the ability to generate ATP from food. In some way the brain detects the shortage of ATP and responds by increasing appetite.

by Tom Stockdale.